Cyclophosphamide induces ovarian granulosa cell ferroptosis via a mechanism associated with HO-1 and ROS-mediated mitochondrial dysfunction

Abstract Abnormal granulosa cell (GC) death contributes to cyclophosphamide (CTX) induced primary ovarian insufficiency (POI).To investigate the contribution of GCs to POI, gene profiles of GCs exposed to CTX were assessed The tragic paradoxical effect of telemedicine on healthcare disparities- a time for redemption: a narrative review using RNA-Se

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